Buy AC-262 536 Sarm Capsules

Description

AC-262 536 Sarm Capsules

Acadia Pharmaceuticals developed AC-262 536 for the treatment of prostate cancer and Alzheimer’s disease. It stimulates muscular growth similarly to testosterone. However, when the compound comes into direct contact with the body’s androgen receptors, muscle growth and strength improve without increasing the risk of prostate cancer, damage to sexual tissue, or liver enzymes.

These Selective Androgen Receptor Modulators have only been tested on animals, so its effectiveness and benefits are not yet determined. However, bodybuilders and athletes use the compound because it’s similar to testosterone but without the harmful side effects.

AC-262 536 is a selective androgen receptor modulator that is non-steroidal (SARM). The initial study on this SARM began in 2007 with animal investigations, and no human trials or studies have been conducted since then. AC-262,536 binds directly to the androgen receptor, decreasing the risk of cancer of the prostate, shrinking the testicles, and changing the sexual tissue.

However, it still helps strengthen bone density and muscle growth.

AC-262536 has demonstrated in animal models that it may be more potent than testosterone at binding receptors and inhibiting luteinizing hormone (LH). Research on the compound AC-262536 found that testosterone could potentially promote muscular growth.

In addition, AC-262 536 may have prostate cancer advantages since it strongly impacts the dihydrotestosterone (DHT) prostate cancer effects in human cancer cells (in vitro).

Specifications

Formula: C18H18N2O
Molar Mass: 278.355 g·mol−1

Size: 60 Capsules | 10mg per capsule

Research

The benefits of AC-262 536 sarm capsules are still being investigated. However, the following benefits have been demonstrated by prior research and other forms of evidence. A few additional advantages may have emerged by the time the present research trials are concluded. The following benefits are supported by animal studies conducted on rodents and anecdotal evidence provided by current medicine users.

• Results in an increase in muscle mass. By boosting anabolic characteristics, the medication can considerably increase muscle size. This makes it equivalent to testosterone but without testosterone’s adverse effects [1].
• May aid in Alzheimer’s disease treatment [2]. As the medicine has the ability to enhance androgen receptor levels, it may help prevent dementia and comparable conditions. Additionally, the medication can improve spatial memory control [3].
• Could help prevent prostate cancer. Due to its antagonistic impact on DHT (dihydrotestosterone), AC-262 may reduce the chance of developing prostate cancer. This is a significant benefit because, according to numerous studies, DHT has historically been linked to the development of prostate cancer due to the increased cell growth in the prostate area [4].
• Reduces levels of luteinizing hormone (LH). Although LH supports natural testosterone creation, it may also serve as a breeding ground for some disorders when LH levels are elevated. Despite the drop in LH levels, there is no significant decrease in the efficacy or strength of AC-262 [5].

Summary

While research on the benefits of AC-262 536 sarm capsules has been conducted on animals, no human trials have been completed. Therefore, the claimed benefits of this chemical cannot be supported. Instead, the vast majority are based on the personal reviews gathered by individuals who have chosen to use the product. Several of these advantages include:

• Increased muscular development
• By counteracting the effects of dihydrotestosterone, the risk of prostate cancer may be reduced.
• Potential restoration of cognitive functions in Alzheimer’s disease patients.
• Enhanced fat loss
• Strengthened bone mass.
• A rise in luteinizing hormone suppression.

References

[1] https://pubmed.ncbi.nlm.nih. gov/18164613/

[2] https://www.ncbi.nlm.nih.gov/ pmc/ articles/PMC3867967/

[3] https://www.researchgate.net/ publication/233513007

[4] https://pubmed.ncbi.nlm.nih. gov/29465427/

[5] https://pubmed.ncbi.nlm.nih. gov/20798389/

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